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首播

重播

  日本研究人员日前宣布,他们弄清了导致胃癌细胞增殖的胃癌干细胞运作机制。这一成果将有助于开发出防止胃癌细胞增殖的药物。

  在胃癌等癌症中,癌干细胞不断分裂是癌细胞增殖的原因,但是迄今为止一直不清楚其运作机制。庆应义塾大学医学部教授佐谷秀行领导的研究小组发现,癌干细胞表面的“CD44”蛋白质发挥了重要作用,它会使能遏制癌细胞增殖的“p38”蛋白质无法发挥作用。

  细胞内存在具有抗氧化作用的物质时,“p38”就会发挥作用,遏制癌细胞的增殖。而“CD44”则会吸收细胞内具有抗氧化作用的物质,从而使“p38”无法发挥作用。

  研究小组在利用患有胃癌的老鼠进行实验时发现,如果遏制了“CD44”的作用,“p38”就开始发挥作用,从而使肿瘤缩小。

  研究小组认为,如果今后能够开发出遏制“CD44”蛋白质的药物,就有可能遏制胃癌以及其他多种癌症细胞的增殖。

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